描述：

它是由Goldspink等人首先在肌肉中发现的。相比之下，在人类肌肉中，一个49个碱基的插入片段改变了机械因子（MGF）的阅读框架。当机械过载被引入肌肉（如通过体重训练）时，基因在身体反应期间被释放并且被差异地拼接。最初，它被拼接以主要产生（称为MGF剪接变体1）。

这种早期剪接刺激了卫星细胞的激活。这又允许激活额外的未损伤的核以生长新的肌肉纤维和组织.MGF的出现也引发新的蛋白质合成的上调。在这种初始剪接进入MGF之后，生产然后转向从肝脏产生1Ea的全身释放，其也上调蛋白质合成。

在肌肉修复的愈合和再生长阶段的过程中，表达剪接变体被认为是身体产生新肌肉的主要合成代谢机制.MGF可作为可注射肽获得，并且已经被轶事显示注射它将导致该区域的反应，导致局部肌肉生长。
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Description:

It was first discovered in the muscle by Goldspink, et al. In human muscle, a 49-base insert changes the reading frame in mechano factor (MGF) as compared. When mechanical overload is introduced to a muscle (as by weight training), the gene released and is differentially spliced during the bodies response. Initially, it it is spliced to produce predominantly (called the MGF splice variant 1).

This early splicing stimulates satellite cells into activation. Which in turn allows the activation of extra undamaged nuclei to grow new muscle fiber and tissue.The appearance of MGF also initiates the upregulation of new protein synthesis. After this initial splicing into MGF, production then switches towards producing a systemic release of 1Ea from the liver, which also upregulates protein synthesis as well.

The expression splice variants, over the course of the healing and regrowth phase of muscle repair is thought to be the primary anabolic mechanism by which the body produces new muscle.MGF is available as an injectable peptide, and it has been anecdotally shown that injecting it will cause a response in the area resulting in localized muscle growth.
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